|
1）Schroder AR, Shinn P, Chen H, et al. HIV-1 integration in the human genome favors active genes and local hotspots. Cell. 2002; 110: 521-9
|
|
|
|
2）Wu X, Li Y, Crise B, et al. Transcription start regions in the human genome are favored targets for MLV integration. Science. 2003; 300: 1749-51
|
|
|
|
|
|
3）Han Y, Lassen K, Monie D, et al. Resting CD4＋ T cells from human immunodeficiency virus type 1 (HIV-1)-infected individuals carry integrated HIV-1 genomes within actively transcribed host genes. J Virol. 2004; 78: 6122-33
|
|
|
|
|
|
4）Pion M, Jordan A, Biancotto A, et al. Transcriptional suppression of in vitro-integrated human immunodeficiency virus type 1 does not correlate with proviral DNA methylation. J Virol. 2003; 77: 4025-32
|
|
|
|
|
|
5）Matsuoka M. Human T-cell leukemia virus type I (HTLV-I) infection and the onset of adult T-cell leukemia (ATL). Retrovirology. 2005; 2: 27
|
|
|
|
|
|
6）Matsuoka M, Jeang KT. Human T-cell leukemia virus type 1 (HTLV-1) infectivity and cellular transformation. Nat Rev Cancer. 2007; 7: 270-80
|
|
|
|
|
|
7）Doi K, Wu X, Taniguchi Y, et al. Preferential selection of human T-cell leukemia virus type I provirus integration sites in leukemic versus carrier states. Blood. 2005; 106: 1048-53
|
|
|
|
|
|
8）Jordan A, Bisgrove D, Verdin E. HIV reproducibly establishes a latent infection after acute infection of T cells in vitro. EMBO J. 2003; 22: 1868-77
|
|
|
|
|
|
9）Taniguchi Y, Nosaka K, Yasunaga J, et al. Silencing of human T-cell leukemia virus type I gene transcription by epigenetic mechanisms. Retrovirology. 2005; 2: 64
|
|
|
|
|
|
10）Koiwa T, Hamano-Usami A, Ishida T, et al. 5'- long terminal repeat-selective CpG methylation of latent human T-cell leukemia virus type 1 provirus in vitro and in vivo. J Virol. 2002; 76: 9389-97
|
|
|
|
|
|
11）Takeda S, Maeda M, Morikawa S, et al. Genetic and epigenetic inactivation of tax gene in adult T-cell leukemia cells. Int J Cancer. 2004; 109: 559-67
|
|
|
|
|
|
12）Satou Y, Yasunaga J-I, Yoshida M, et al. The HTLV-I bZIP factor gene mRNA supports proliferation of adult T-cell leukemia cells. Proc Natl Acad Sci USA. 2006; 103: 720-5
|
|
|
|
|
|
13）Siliciano JD, Kajdas J, Finzi D, et al. Long-term follow-up studies confirm the stability of the latent reservoir for HIV-1 in resting CD4＋ T cells. Nat Med. 2003; 9: 727-8
|
|
|
|
|
|
14）Marcello A. Latency: the hidden HIV-1 challenge. Retrovirology. 2006; 3: 7
|
|
|
|
|
|
15）Lehrman G, Hogue IB, Palmer S, et al. Depletion of latent HIV-1 infection in vivo: a proof-of-concept study. Lancet. 2005; 366: 549-55
|
|
|
|
|
|
16）Tajima S, Ikawa Y, Aida Y. Complete bovine leukemia virus (BLV) provirus is conserved in BLV-infected cattle throughout the course of B-cell lymphosarcoma development. J Virol. 1998; 72: 7569-76
|
|
|
|
|
|
17）Tajima S, Tsukamoto M, Aida Y. Latency of viral expression in vivo is not related to CpG methylation in the U3 region and part of the R region of the long terminal repeat of bovine leukemia virus. J Virol. 2003; 77: 4423-30
|
|
|
|
|
|
18）Achachi A, Florins A, Gillet N, et al. Valproate activates bovine leukemia virus gene expression, triggers apoptosis, and induces leukemia/lymphoma regression in vivo. Proc Natl Acad Sci USA. 2005; 102: 10309-14
|
|
|
|
|
|
19）Vine AM, Heaps AG, Kaftantzi L, et al. The role of CTLs in persistent viral infection: cytolytic gene expression in CD8＋ lymphocytes distinguishes between individuals with a high or low proviral load of human T cell lymphotropic virus type 1. J Immunol. 2004; 173: 5121-9
|
|
|
|
|
|
20）Lezin A, Olindo S, Verlaeten O, et al. One year follow up of proviral loads and tax gene expression in 19 HAM/TSP patients treated with Valproate. AIDS Res Hum Retroviruses. 2007; 23: 594
|
|
|
|
|