医中誌リンクサービス


文献リスト

1)Nakae S, Komiyama Y, Yokoyama H, et al. IL-1 is required for allergen-specific Th2 cell activation and the development of airway hypersensitivity response. Int Immunol. 2003; 15(4): 483-90
PubMed CrossRef
医中誌リンクサービス
2)Schmitz N, Kurrer M, Kopf M. The IL-1 receptor 1 is critical for Th2 cell type airway immune responses in a mild but not in a more severe asthma model. Eur J Immunol. 2003; 33(4): 991-1000
PubMed CrossRef
医中誌リンクサービス
3)Ewart SL, Kuperman D, Schadt E, et al. Quantitative trait loci controlling allergen-induced airway hyperresponsiveness in inbred mice. Am J Respir Cell Mol Biol. 2000; 23(4): 537-45
PubMed
医中誌リンクサービス
4)Yoshimoto T, Tsutsui H, Tominaga K, et al. IL-18, although antiallergic when administered with IL-12, stimulates IL-4 and histamine release by basophils. Proc Natl Acad Sci U S A. 1999; 96(24): 13962-6
PubMed CrossRef
医中誌リンクサービス
5)Yoshimoto T, Mizutani H, Tsutsui H, et al. IL-18 induction of IgE: dependence on CD4+ T cells, IL-4 and STAT6. Nat Immunol. 2000; 1(2): 132-7
PubMed CrossRef
医中誌リンクサービス
6)Black RA, Kronheim SR, Merriam JE, et al. A pre-aspartate-specific protease from human leukocytes that cleaves pro-interleukin-1 beta. J Biol Chem. 1989; 264(10): 5323-6
PubMed
医中誌リンクサービス
7)Cerretti DP, Kozlosky CJ, Mosley B, et al. Molecular cloning of the interleukin-1 beta converting enzyme. Science. 1992; 256(5053): 97-100
PubMed
医中誌リンクサービス
8)Thornberry NA, Bull HG, Calaycay JR, et al. A novel heterodimeric cysteine protease is required for interleukin-1 beta processing in monocytes.Nature. 1992; 356(6372): 768-74
PubMed
医中誌リンクサービス
9)Hofmann K, Bucher P, Tschopp J. The CARD domain: a new apoptotic signalling motif. Trends Biochem Sci. 1997; 22(5): 155-6
PubMed CrossRef
医中誌リンクサービス
10)Martinon F, Burns K, Tschopp J. The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta. Mol Cell. 2002; 10(2): 417-26
PubMed CrossRef
医中誌リンクサービス
11)Agostini L, Martinon F, Burns K, et al. NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoin­flammatory disorder. Immunity. 2004; 20(3): 319-25
PubMed CrossRef
医中誌リンクサービス
12)Mariathasan S, Weiss DS, Newton K, et al. Cryopyrin activates the inflammasome in response to toxins and ATP. Nature. 2006; 440(7081): 228-32
PubMed
医中誌リンクサービス
13)Lamkanfi M, Dixit VM. Inflammasomes: guardians of cytosolic sanctity. Immunol Rev. 2009; 227(1): 95-105
PubMed CrossRef
医中誌リンクサービス
14)Ghayur T, Banerjee S, Hugunin M, et al. Caspase-1 processes IFN-gamma-inducing factor and regulates LPS-induced IFN-gamma production. Nature. 1997; 386(6625): 619-23
PubMed
医中誌リンクサービス
15)Talabot-Ayer D, Lamacchia C, Gabay C, et al. Interleukin-33 is biologically active indepen­dently of caspase-1 cleavage. J Biol Chem. 2009; 284(29): 19420-6
PubMed CrossRef
医中誌リンクサービス
16)Hoffman HM, Mueller JL, Broide DH, et al. Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoin­flammatory syndrome and Muckle-Wells syndrome. Nat Genet. 2001; 29(3): 301-5
PubMed CrossRef
医中誌リンクサービス
17)Feldmann J, Prieur AM, Quartier P, et al. Chronic infantile neurological cutaneous and articular syndrome is caused by mutations in CIAS1, a gene highly expressed in polymorphonuclear cells and chondrocytes. Am J Hum Genet. 2002; 71(1): 198-203
PubMed CrossRef
医中誌リンクサービス
18)French FMF Consortium. A candidate gene for familial Mediterranean fever. Nat Genet. 1997; 17(1): 25-31
PubMed CrossRef
医中誌リンクサービス
19)Richards N, Schaner P, Diaz A, et al. Interaction between pyrin and the apoptotic speck protein (ASC) modulates ASC-induced apoptosis. J Biol Chem. 2001; 276(42): 39320-9
PubMed CrossRef
医中誌リンクサービス
20)Yu JR, Leslie KS. Cryopyrin-associated periodic syndrome: an update on diagnosis and treatment response. Curr Allergy Asthma Rep. 2011; 11(1): 12-20
PubMed CrossRef
医中誌リンクサービス
21)Hitomi Y, Ebisawa M, Tomikawa M, et al. Associations of functional NLRP3 polymorphisms with susceptibility to food-induced anaphylaxis and aspirin-induced asthma. J Allergy Clin Immunol. 2009; 124(4): 779-85 e776
PubMed CrossRef
医中誌リンクサービス
22)Brickey WJ, Alexis NE, Hernandez ML, et al. Sputum inflammatory cells from patients with allergic rhinitis and asthma have decreased inflammasome gene expression. J Allergy Clin Immunol. 2011; 128(4): 900-3
PubMed CrossRef
医中誌リンクサービス
23)Eisenbarth SC, Colegio OR, O’Connor W, et al. Crucial role for the Nalp3 inflammasome in the immunostimulatory properties of aluminium adjuvants. Nature. 2008; 453(7198): 1122-6
PubMed
医中誌リンクサービス
24)Franchi L, Núñez G. The Nlrp3 inflammasome is critical for aluminium hydroxide-mediated IL-1beta secretion but dispensable for adjuvant activity. Eur J Immunol. 2008; 38(8): 2085-9
PubMed CrossRef
医中誌リンクサービス
25)Besnard AG, Guillou N, Tschopp J, et al. NLRP3 inflammasome is required in murine asthma in the absence of aluminum adjuvant. Allergy. 2011; 66(8): 1047-57
PubMed CrossRef
医中誌リンクサービス
26)Ather JL, Ckless K, Martin R, et al. Serum amyloid A activates the NLRP3 inflammasome and promotes Th17 allergic asthma in mice. J Immunol. 2011; 187(1): 64-73
PubMed CrossRef
医中誌リンクサービス
27)Kool M, Willart MA, van Nimwegen M, et al. An unexpected role for uric acid as an inducer of T helper 2 cell immunity to inhaled antigens and inflammatory mediator of allergic asthma.Immunity. 2011; 34(4): 527-40
PubMed CrossRef
医中誌リンクサービス
28)Allen IC, Jania CM, Wilson JE, et al. Analysis of NLRP3 in the development of allergic airway disease in mice. J Immunol. 2012; 188(6): 2884-93
PubMed CrossRef
医中誌リンクサービス


NPO医学中央雑誌刊行会
https://www.jamas.or.jp/
info@jamas.or.jp