医中誌リンクサービス


文献リスト

1)Ju BG, Lunyak VV, Perissi V, et al. A topoisomerase IIbeta-mediated dsDNA break required for regulated transcription. Science. 2006; 312: 1798-802
PubMed CrossRef
医中誌リンクサービス
2)Suberbielle E, Sanchez PE, Kravitz AV, et al. Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β. Nat Neurosci. 2013; 16: 613-21
PubMed CrossRef
医中誌リンクサービス
3)Cirrito JR, Yamada KA, Finn MB, et al. Synaptic activity regulates interstitial fluid amyloid-beta levels in vivo. Neuron. 2005; 48: 913-22
PubMed CrossRef
医中誌リンクサービス
4)Cirrito JR, Kang JE, Lee J, et al. Endocytosis is required for synaptic activity-dependent release of amyloid-beta in vivo. Neuron. 2008; 58: 42-51
PubMed CrossRef
医中誌リンクサービス
5)Roberson ED, Scearce-Levie K, Palop JJ, et al. Reducing endogenous tau ameliorates amyloid beta-induced deficits in an Alzheimerʼs disease mouse model. Science. 2007; 316: 750-4
PubMed CrossRef
医中誌リンクサービス
6)Roberson ED, Halabisky B, Yoo JW, et al. Amyloid-β/Fyn-induced synaptic, network, and cognitive impairments depend on tau levels in multiple mouse models of Alzheimerʼs disease. J Neurosci. 2011; 31: 700-11
PubMed CrossRef
医中誌リンクサービス
7)Cissé M, Halabisky B, Harris J, et al. Reversing EphB2 depletion rescues cognitive functions in Alzheimer model. Nature. 2011; 469: 47-52
PubMed CrossRef
医中誌リンクサービス
8)Jin M, Shepardson N, Yang T, et al. Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration. Proc Natl Acad Sci U S A. 2011; 108: 5819-24
PubMed CrossRef
医中誌リンクサービス
9)Sanders DW, Kaufman SK, DeVos SL, et al. Distinct tau prion strains propagate in cells and mice and define different tauopathies. Neuron. 2014; 82: 1271-88
PubMed CrossRef
医中誌リンクサービス
10)La Spada AR, Wilson EM, Lubahn DB, et al. Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy. Nature. 1991; 352: 77-9
PubMed CrossRef
医中誌リンクサービス
11)Orr HT, Chung MY, Banfi S, et al. Expansion of an unstable trinucleotide CAG repeat in spino-cerebellar ataxia type 1. Nat Genet. 1993; 4: 221-6
PubMed CrossRef
医中誌リンクサービス
12)A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntingtonʼs disease chromosomes. The Huntingtonʼs Disease Collaborative Research Group. Cell. 1993; 72: 971-83
PubMed CrossRef
医中誌リンクサービス
13)Imafuku I, Waragai M, Takeuchi S, et al. Polar amino acid-rich sequences bind to polyglutamine tracts. Biochem Biophys Res Commun. 1998; 253: 16-20
PubMed CrossRef
医中誌リンクサービス
14)Hirabayashi M, Inoue K, Tanaka K, et al. VCP/p97 in abnormal protein aggregates, cytoplasmic vacuoles, and cell death, phenotypes relevant to neurodegeneration. Cell Death Differ. 2001; 8: 977-84
PubMed CrossRef
医中誌リンクサービス
15)Fujita K1, Nakamura Y, Oka T, A functional deficiency of TERA/VCP/p97 contributes to impaired DNA repair in multiple polyglutamine diseases. Nat Commun. 2013; 4: 1816
PubMed
医中誌リンクサービス
16)Bartolome F, Wu HC, Burchell VS, et al. Pathogenic VCP mutations induce mitochondrial uncoupling and reduced ATP levels. Neuron. 2013; 78: 57-64
PubMed CrossRef
医中誌リンクサービス
17)Qi ML, Tagawa K, Enokido Y, et al. Proteome analysis of soluble nuclear proteins reveals that HMGB1/2 suppress genotoxic stress in polyglutamine diseases. Nat Cell Biol. 2007; 9: 402-14
PubMed CrossRef
医中誌リンクサービス
18)Enokido Y, Tamura T, Ito H, et al. Mutant huntingtin impairs Ku70-mediated DNA repair. J Cell Biol. 2010; 189: 425-43
PubMed CrossRef
医中誌リンクサービス
19)Barclay SS, Tamura T, Ito H, et al. Systems biology analysis of Drosophila in vivo screen data elucidates core networks for DNA damage repair in SCA1. Hum Mol Genet. 2014; 23:1345-64
PubMed CrossRef
医中誌リンクサービス


NPO医学中央雑誌刊行会
https://www.jamas.or.jp/
info@jamas.or.jp