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10)Hossain GS, van Thienen JV, Werstuck GH, et al. TDAG51 is induced by homocysteine, promotes detachment-mediated programmed cell death, and contributes to the cevelopment of atherosclerosis in hyperhomocysteinemia. J Biol Chem. 2003; 278: 30317-27
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19)Zhou AX, Wang X, Lin CS, et al. C/EBP-homologous protein (CHOP) in vascular smooth muscle cells regulates their proliferation in aortic explants and atherosclerotic lesions. Circ Res. 2015; 116: 1736-43
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20)Tabas I. The role of endoplasmic reticulum stress in the progression of atherosclerosis. Circ Res. 2010; 107: 839-50
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21)Feng B, Yao PM, Li Y, et al. The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages. Nat Cell Biol. 2003; 5: 781-92
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25)Seimon T, Liao X, Magallon J, et al. Atherogenic lipids and lipoproteins trigger CD36-TLR2-dependent apoptosis in macrophages undergoing endoplasmic reticulum stress. Cell Metab. 2010; 12: 467-82
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26)Li F, Guo Y, Sun S, et al. Free cholesterol-induced macrophage apoptosis is mediated by inositol-requiring enzyme 1 alpha-regulated activation of Jun N-terminal kinase. Acta Biochim Biophys Sin (Shanghai). 2008; 40: 226-34
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33)Miyazaki-Anzai S, Masuda M, Demos-Davies KM, et al. Endoplasmic reticulum stress effector CCAAT/enhancer-binding protein homologous protein (CHOP) regulates chronic kidney disease-induced vascular calcification. J Am Heart Assoc. 2014; 3: e000949
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34)Dorweiler B, Grechowa I, Wallrath A, et al. Activation of the proapoptotic unfolded protein response in plaques of the human carotid artery. Eur J Vasc Endovasc Surg. 2014; 48: 248-57
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35)Ishimura S, Furuhashi M, Mita T, et al. Reduction of endoplasmic reticulum stress inhibits neointima formation after vascular injury. Sci Rep. 2014; 4: 6943
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